Cytochrome C is extracted from the heart of pigs or cattle and is an electron transporter in the process of biooxidation. The effect is similar to that of coenzyme. When the tissue is hypoxic, the cell permeability is increased. After the injection, cytochrome C can enter the cell to correct the cell respiration and promote the metabolism of the substance.
This product is a cell respiratory activating enzyme, as a first-aid drug and ancillary drugs for tissue hypoxia treatment, such as carbon monoxide poisoning, sleeping pills poisoning, primary child suspended animation, irreversible shock hypoxia, pre-anesthesia treatment; pneumonia, lung cancer, silicosis, lung Breathing, dyspnea caused by bronchiectasis, hypoxia of alpine disease, cerebral vascular disorders, brain trauma, cerebral hemorrhage, cerebral arteriosclerosis, sequelae of concussion, sequela of Japanese encephalitis, pertussis encephalopathy, etc. Toxic myocardial damage, myocarditis, myocardial insufficiency, angina pectoris, diphtheria myocarditis, myocardial infarction, etc.; reduction of white blood cells caused by anticancer drugs, limb circulatory disorders, nerve palsy, liver disease, nephritis also have an improving effect.
Studies on apoptosis have shown that cytochrome C is involved in apoptosis, and cytochrome C leaking from mitochondria induces apoptosis.
Pharmacological effects of cytochrome C
